Buy Cardiovascular Drugs Discussion

Buy Cardiovascular Drugs Discussion
Buy Cardiovascular Drugs Discussion
CHOOSE THE MOST APPROPRIATE RESPONSE
34.1 Under physiological conditions the rate limiting enzyme
in the generation of angiotensin II is:
A. Renin
B. Angiotensin converting enzyme
C. Aminopeptidase
D. Angiotensinase (p. 445)
34.2 Angiotensin II causes rise in blood pressure by:
A. Direct vasoconstriction
B. Releasing adrenaline from adrenal medulla
C. Increasing central sympathetic tone
D. All of the above (p. 445-446)
34.3 Angiotensin III is equipotent to angiotensin II in:
A. Releasing aldosterone from adrenal cortex
B. Promoting Na and water reabsorption by
direct intrarenal action
C. Causing vasoconstriction
D. Contracting intestinal smooth muscle
(p. 445, 446)
Buy Cardiovascular Drugs Discussion
34.4 The following is a pressor peptide that can be
generated both in circulation as well as locally in
certain tissues:
A. Bradykinin
B. Angiotensin
C. Kallidin
D. Plasmin (p. 445, 454)
34.1 A 34.2 D 34.3 A 34.4 B
218 MCQs in Pharmacology 218
34.5 The following factors enhance renin release from the
kidney except:
A. Fall in blood pressure
B. Reduction in blood volume
C. Enhanced sympathetic activity
D. Volume overload (p. 446, 447)
34.6 Angiotensin II plays a key role in the following risk
factor for ischaemic heart disease:
A. Hypercholesterolemia
B. Ventricular hypertrophy
C. Carbohydrate intolerance
D. Cardiac arrhythmia (p. 446)
34.7 Ventricular remodeling after myocardial infarction
involves the mediation of:
A. Angiotensin II
B. Prostaglandin
C. Bradykinin
D. Thromboxane A2 (p. 446)
34.8 Captopril pretreatment:
A. Inhibits the pressor action of angiotensin I
B. Inhibits the pressor action of angiotensin II
C. Potentiates the depressor action of bradykinin
D. Both ‘A’ and ‘C’ are correct (p. 449)
34.9 Captopril produces greater fall in blood pressure in:
A. Diuretic treated patients
B. Patients having low plasma renin activity
C. Sodium replete normotensive individuals
D. Untreated CHF patients (p. 449)
34.5 D 34.6 B 34.7 A 34.8 D 34.9 A
Cardiovascular Drugs 219
Buy Cardiovascular Drugs Discussion
34.10 Potentiation of bradykinin appears to play a role in
the following effects of angiotensin converting
enzyme inhibitors except:
A. Fall in BP in the short term
B. Fall in BP in the long term
C. Cough in susceptible individuals
D. Angioedema in susceptible individuals
(p. 449)
34.11 Enalapril differs from captopril in that:
A. It blocks angiotensin II receptors
B. It does not produce cough as a side effect
C. It is less liable to cause abrupt first dose
hypotension
D. It has a shorter duration of action
(p. 450, 451)
34.12 Enalapril differs from captopril in the following
features except:
A. It is dose to dose more potent
B. Its oral absorption is not affected by food in
stomach
C. It acts more rapidly
D. It has longer duration of action (p. 450, 451)
34.13 The following angiotensin converting enzyme inhibitor can reduce cardiac contractility:
A. Captopril
B. Enalapril
C. Perindopril
D. Lisinopril (p. 451)
34.10 B 34.11 C 34.12 C 34.13 D
220 MCQs in Pharmacology 220
34.14 Advantages of angiotensin converting enzyme inhibitors as antihypertensive include the following
except:
A. They tend to reverse left ventricular hypertrophy
B. Their efficacy is not reduced by nonsteroidal
antiinflammatory drugs
C. They do not worsen blood lipid profile
D. They do not impair work performance
(p. 450, 451)
34.15 The following drug increases cardiac output in
congestive heart failure without having any direct
myocardial action:
A. Captopril
B. Digoxin
C. Amrinone
D. Dobutamine (p. 452, 469)
34.16 Angiotensin converting enzyme inhibitors reduce
the following haemodynamic parameters in congestive heart failure except:
A. Systemic vascular resistance
B. Right atrial pressure
C. Cardiac output
D. Heart rate × pressure product (p. 452)
34.17 Angiotensin converting enzyme inhibitors afford
maximum protection against progression of heart
failure when used:
A. At the higher therapeutic dose range over
long term
B. At the maximum tolerated dose only till
haemodynamic compensation is restored
C. At low doses over long term
D. At low doses along with diuretics/digoxin
(p. 452)
34.14 B 34.15 A 34.16 C 34.17 A
Cardiovascular Drugs 221
34.18 In post-myocardial infarction and other high cardiovascular risk subjects but without hypertension or
heart failure, prolonged ACE inhibitor medication
has been found to:
A. Increase the chances of sudden cardiac death
B. Reduce the incidence of fatal as well as nonfatal myocardial infarction or stroke
C. Lower the risk of developing heart failure
and diabetes
D. Both ‘B‘ and ‘C’ (p. 452)
34.19 Which of the following statements most closely
describes the current status of angiotensin converting enzyme inhibitors in congestive heart failure:
A. They are the first choice drugs unless contraindicated
B. They are used when diuretics alone fail
C. They are a substitute for digitalis
D. They are to be used as adjuncts only in
resistant cases (p. 452)
34.20 Long term ACE inhibitor therapy may retard the
progression of:
A. Diabetic nephropathy
B. Diabetic retinopathy
C. Hypertensive nephropathy
D. All of the above (p. 452-453)
34.21 The following drug has been demonstrated to retard
progression of left ventricular dysfunction and
prolong survival of congestive heart failure patients:
A. Digoxin
B. Furosemide
C. Enalapril
D. Amrinone (p. 452, 469)
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